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angiew wrote:Hi Davids1,
Any indicators of pro-oxidant effect of such high dosage?
Countless biochemists and chemists discovered what in the wall of the artery causes Lp(a) to adhere and form atherosclerotic plaques and ultimately lead to heart disease, strokes, and peripheral arterial disease. The answer is that there is a particular amino acid in a protein in the wall of the artery - lysine - which is one of the twenty amino acids that binds the Lp(a) and causes atherosclerotic plaques to develop. “I THINK IT IS A VERY IMPORTANT DISCOVERY."
Symbol: lys k
Molecular formula: C6H14N2O2
Molecular weight: 146.19
Isoelectric point (pH): 9.59
Lysine is an amino acid that is not produced by the body, but is essential for the growth and production of proteins. Lysine is classified, as one of the nine essential amino acids in that is must be obtained from outside sources such as foods and supplements. It is required for growth and bone development in children, assists in calcium absorption and maintaining the correct nitrogen balance in the body and maintaining lean body mass. It is also fundamental for the production of antibodies, enzymes, hormones, and for tissue growth and repair. Most individuals have an adequate intake of lysine; however lysine levels may be low in vegetarians and low-fat dieters. Without enough lysine or any other of the eight essential amino acids, the body cannot build protein to sustain muscle tissue.
The answer is that there is a particular amino acid in a protein in the wall of the artery - lysine -
Lysine is an amino acid that is not produced by the body, but is essential for the growth and production of proteins. Lysine is classified, as one of the nine essential amino acids in that is must be obtained from outside sources such as foods and supplements.
Johnwen wrote:Wouldn’t it be absorbed by the cells before it caused the LP(a) to become attached to it??
Johnwen wrote:Wouldn’t supplementing with L-Lysine be more detrimental then beneficial since taking in more lysine would cause more attachments to these said proteins within the arterial system which would cause more blockages, which would make it a causative agent rather then a correcting agent??
randian wrote:Doesn't the Pauling theory imply that Lp(a) is attaching itself to free lysine, not lysine bound up in arterial walls?
Owen/Pauling wrote:The answer is that there is a particular amino acid in a protein in the wall of the artery - lysine -
IMO, you ask an intelligent question.How has your health been since then?
I have not been to a doctor since 1971 [when I was in college and it was free (when I burnt myself with some hot popcorn oil)], so I would not know anything about my "blood work." Up until I started with Bowel Tolerance dosing of ascorbic acid, although my health was [relatively] quite good [I exercised fairly extensively], I still did get an [approximately] annual "cold" or so [certainly more extensive, i.e. somewhat more severe and definitely longer lasting, than anything that I have had in the past 19 years].What about blood work and medical exams, any problems?
This question surprises me, Angiew. Each molecule of ascorbic acid has two "extra" electrons. These electrons are, by definition, reducing, i.e. anti-oxidixing. Like Owen wrote, if the body needs the ascorbate to perform "oxidant" type activities, it simply takes and/or makes what it wants, e.g. dehydroascorbate as a virucidal, etc., etc.Any indicators of pro-oxidant effect of such high dosage?
In other words, Pauling himself, as far as I can tell, did not use Bowel Tolerance as his way of deciding how much ascorbate he [himself] should ingest, i.e. throughout the day. As you can probably tell, I believe that is an error for anyone, e.g. as Pauling got older, and subsequently contracted cancer, I assume his Bowel Tolerance would have been increasing. In fact, I do not know for sure that Cathcart himself used Bowel Tolerance as his means of deciding how much he should ingest when he felt well.Pauling never mentioned using Bowel Tolerance as his means of deciding upon dosage amount.
I do not understand. If I "tend to retain mercury" why would it not show up in my hair?What does having a lot of mercury in your mouth but none in your hair tell you!?!! That you are a low mercury excretor (same problem with autistics). You tend to retain mercury...
By this do you mean that because I am a [naturally] "low mercury excretor" I need to ingest more ascorbate to facilitate that excretion?...so your high dosages of ascorbate have a purpose!
OWEN wrote: So what do you think I said that Pauling said that didn't make sense?
OWEN wrote: The answer is that there is a particular amino acid in a protein in the wall of the artery - lysine -
WIKI wrote:Allysine is a derivative of lysine, used in the production of elastin and collagen. It is produced by the actions of the enzyme lysyl oxidase on lysine in the extracellular matrix and is essential in the crosslink formation that stabilizes collagen and elastin.
I assume you meant milligrams [vs. "grams"]. It would be interesting to see the science on that particular theory. I doubt it's true, but if the pharma/medico "mafia" wants to pay for enough "studies" I have little doubt they can convince someone to "produce" the results they are after!...I've read many places online that state (not studies but people's 2 cents) that vit c is pro-oxidant over 2500+ grams.
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