Vitamin C Forum

Posting Nik2201's lipids (heterozygous familial hypercholesterolemia), who is undergoing my developed ASO protocol. The test was carried out before ASO, like the earlier ones posted. Would repeat ALL after ASO protocol, APO-A is great and B not that bad either, hoping once repeated after ASO would be as great as was back in '22.





APO-A1 approx. the same, APO-B has doubled...... no issues would hopefully be fixed.

Why am so concerned about normalizing Total Cholesterol etc., since it is the reason/cause for his Crohn's.

Atrial Fibrillation etc. were cured just need to be careful not to disrupt the equilibrium. Like posted before I am treating him like walking on a thin rope, haven't treated all 4/5 conditions in a single person in 37 yrs and that too online.

And haven't witnessed any patient as cooperative as Nik2201 (Btw- I don't consider him as a patient or a member but a great friend), who puts in 200% to my recommendations, with no whining.

Shall request him to repeat all labs after ASO, which shall post.

eDOC!!


PS:
Spent +14 yrs. on a single drug treating humans, could probably have acquired 2 more board certifications.
My personal opinion conv. med is merely palliative, apart from labs.
This and Nik's earlier A-Fib thread is the way to treat and cure a person.
I'm again confused about my specialty since am a conventional neurologist. People are used to specific labels, when I tell them most get confused how a neuro. can treat liver, cardio, endo, renal, autoimmune, ophthalmic, derm, obs/gyn disorders.. list is endless. I probably need to label myself?

All labs post ASO, as I receive. Btw- I don't like the levels (approx. 4 mg/dL) and was expecting better, (around 3 mg/dL) so I asked him to have another ASO injection and redo all labs.



eDOC!!

Performing all required labs relating to his multiple med. conditions, to check all are well balanced, healed and are in a state of equilibrium.





eDOC!!

For a person suffering from familial heterozygous hypercholesterolemia, T. Cholesterol down to 5.8 from 7.2 mmol/L or 224 mg/dL isn't that bad.
LDH down to 3.9 from earlier 4.9.

I probably need to further fine tune my ASO protocol and do some more gene tweaking....

eDOC!!

Hoping and working on to develop ASO to provide a gene knock down as good as this......

https://vitamincfoundation.com/forum/vi ... 1&start=15

eDOC!!

2 reasons that Nik2201 keeps developing new disorders, after the previous 4 got cured.

1. He already has 2 genetic mutations/disorders.

2. In the process of getting those 2/4 cured, his body has started to develop retrotransposons.

The ultimate solution to cure all these 4/5 disorders and NOT to develop any more is to put him on RT blocking agent/drug etc. Else he would keep developing new rare disorders and cure journey might be unending.

eDOC!!

HISTORY
SYSTEM SPECIFIC HISTORY
The history of atrial fibrillation noted.
GENERAL HISTORY
The patient reported no symptoms.
TECHNIQUE
A time-out was observed to confirm the correct patient, procedure, and site; verbal
consent was obtained.
Grayscale and colour Doppler ultrasound examination of the carotid and vertebral artery
system was undertaken on the right and left.
FINDINGS IN DETAIL
CAROTID VESSELS
RIGHT
CAROTID FINDINGS (Right)
Peak right common carotid artery velocity 70 cm/s.
Peak right Internal carotid artery velocity 67 cm/s.
Peak right external carotid artery velocity 108 cm/s.
The visualised right common and internal carotid artery exhibits normal vessel calibre,
waveforms and velocities in the normal range.
There is antegrade flow in the right vertebral artery.
Peak right vertebral artery velocity 28 cm/s.
No focal plaque is identified in the right carotid circulation.
The right carotid intima-media thickness (CMIT) of ~0.52 mm is within the normal range
(at age 56 years, mean CMIT is 0.70 mm. The risk of adverse cardiovascular events is
increased where the CMIT thickness exceeds 0.80 mm).
LEFT
CAROTID FINDINGS (Left)
Peak left common carotid artery velocity 84 cm/s.
Peak left Internal carotid artery velocity 88 cm/s.
Peak left external carotid artery velocity 81 cm/s.
The visualised left common and internal carotid artery exhibits normal vessel calibre,
waveforms and velocities in the normal range.
There is antegrade flow in the left vertebral artery.

Peak left vertebral artery velocity 37 cm/s.
The left carotid intima-media thickness (CMIT) of ~0.71 mm is in the normal range (at age
56 years, mean CMIT is 0.70 mm. The risk of adverse cardiovascular events is increased
where the CMIT thickness exceeds 0.80 mm).
There are focal calcified plaques in the left carotid bulb.

PS: Would probably need to merge the vascular etc. results with Crohn's since are linked and want all his multiple issues to get resolved once and for all.

eDOC!!


PS: https://vitamincfoundation.com/forum/vi ... 1&start=15
Compare with one carried out 3 yrs back, almost similar.

It's NOT a classical ANCA, rather atypical ANCA results, with both nuclear and cytoplasmic Abs. Abs against PR3 fueling his other ailments. A mixed results for Small vessel vasculitis and Crohn's.
Intensity +1, isn't that high, rather low for a patient but the appearance of Abs against PR3 isn't a good sign.

eDOC!!

Repeat ANCA carried in Oct' before starting the protocol:



Note: Compare with the post #2, both MPO and PR3 have almost disappeared......

eDOC!!

Far better after treatment but still is positive. But the intensity vanished, dilution titration mild, and most important Abs against PR3 dropped from 4.0 to 0.3.

Lets see what we get this year or in 2026.

eDOC!!

https://welltica.dk/krop/24-aarig-indla ... 9-vaccine/

https://vitamincfoundation.com/forum/vi ... hp?t=15471

A quite similar case to Nik2201, with minor difference. In his case the culprit causing myocarditis was post recovery SARS-CoV-2 infection, in this link was live mRNA.
Common in both is a dysregulated immune response system unable to clear the virus, mRNA remnants, allowing auto Abs creation against the host. Autoimmune triggering....

eDOC!!

CT ABDOMEN NON CONTRAST
KEY FINDINGS
The liver volume is enlarged for age and gender.
The pancreas mean attenuation is estimated at 30 HU (normal range for a male aged 56
is 31 to 75 HU).
The right kidney volume is estimated at 247 cc (normal range for a male aged 56 is 122 to
234 cc).
The right and left renal parenchymal volume estimate is increased for age and gender.
There is no hydronephrosis or hydroureter on the right and left.
Small bowel loops appear normal in size and wall thickness.
No bowel related mass lesion is identified.
The prostate dimensions are approximately 40 (SI) x 38 (AP) x 50 (RL) mm with a volume
estimate of 40 ml.
IMPRESSION AND RECOMMENDATIONS
Pancreatic attenuation is compatible with minor pancreatic lipomatosis.
The left kidney volume is estimated at 278 cc (normal range for a male aged 56 is 128 to
242 cc).
The cause of renal enlargement is not established.
There are no signs of inflammatory bowel disease identified.
The prostate is enlarged.
Follow up ultrasound of the kidneys in ~6 months could be performed if required
COMMENT
The adult male mean renal length is 112 mm; the normal renal length range varies from
99 mm to 126 mm.
Bilateral nephromegaly may be associated with obesity, Type 2 diabetes mellitus and/or
hyperinsulinaemia.
Correlation with laboratory and clinical features may exclude a diagnosis of acutnephritis.
A prostate volume of 35 cc or more is above the normal range for an adult male patient
aged 51-60.
For a prostate volume of ~40 ml, the upper limit for normal PSA is 2.8 ng/ml (normal
Prostatic Density Index < 0.07 ng/ml/cc).
The PSA may be elevated in conditions including prostate inflammation, bacterial
prostatitis, or urinary tract infection.

Any idea why the liver, kidney and prostate volumes are enlarged?

eDOC!!

The reason for his enlarged organs, is dysregulated immune system responses which are in a state of self destruction, pathological inflammation requiring a flip to a state of repair, else he could possibly go into a multi organ dysfunction.

eDOC!!