Moderator: ofonorow
ofonorow wrote:[b][color=#000080]Thank you for the link/paper. Dr. Levy includes a chapter in STOP AMERICA'S #1 KILLER devoted to Chondroitin Sulfate, which is where I first learned of its value for heart disease.
johnjackson wrote:is that worth adding to the PT ?
pamojja wrote:johnjackson wrote:is that worth adding to the PT ?
Here an article of an old clinical experience with chondroitin sulfate: http://knowledgeofhealth.com/the-man-wh ... rol-drugs/
If its worth adding depends if PT alone is working for you. The only way to know for sure are repeated CAC scores, or ultrasonography of max. CIMT, or arterial examination in the cath lab. All other blood-markers including Lp(a) are only 'risk-markers', which only mean you're at somewhat higher risk, but don's show if plaque growth is progressing or regressing at all.
So without taking at least one of these imaging tests, one can't know Pauling therapy is really working. Unless debilitating symptoms, like angina pains, walking pain, etc. is indeed remedied with Pauling therapy right away.
johnjackson wrote:she'll get retested in october and if the Lp(a) is lowered, then PT is working, if it is not, does that mean PT is not working?
pamojja wrote:
A low or high Lp(a) only shows the 'risk' is different, it doesn't show if plaque is regressing or progressing. Only imaging does.
johnjackson wrote:but he is at low risk for a heart attack, even without imaging?
https://drmalcolmkendrick.org/2018/11/27/what-causes-heart-disease-part-59/
There are many, many, things that can do this. Here is a list. It is non-exhaustive, it is in no particular order, but it may give you some idea of the number of things that can cause CVD, by accelerating endothelial damage:
Smoking
Systemic Lupus Erythematosus
Use of oral steroids
Cushing’s disease
Kawasaki’s disease
Rheumatoid arthritis
High blood pressure
Omeprazole
Avastin
Thalidomide
Air pollution
Lead (the heavy metal)
Mercury
High blood sugar
Erythema nodosum
Rheumatoid arthritis
Low albumin
Acute physical stress
Acute mental stress
Chronic negative mental stress
Chronic Kidney Disease
Dehydration
Sickle cell disease
Malaria
Diabetes/high blood sugar level
Bacterial infections
Viral infections
Vitamin C deficiency
Vitamin B deficiency
High homocysteine level
Chronic kidney disease
Acute renal failure
Cocaine
Angiotensin II
Activation of the renin aldosterone angiotensin system (RAAS) etc.
Blimey, yes, that list was just off the top of my head, I could get you another fifty without much effort. And no, I did not just make it up. I have studied every single one of those factors, and many more, in exhaustive detail. The extent of how many factors there are, should not really come as a surprise to anyone, but it usually does.
johnjackson wrote:imaging only shows if plaque is changing?
johnjackson wrote:but what will change my PT ? nothing, right?
pamojja wrote:johnjackson wrote:but he is at low risk for a heart attack, even without imaging?
That would be the case if Lp(a) would be the only risk factor. Sadly, there are too many things which can damage the endothelium and start the repair process of plaque-growth. Here a list of factors by a very knowledgeable cholesterol-skeptic:
johnjackson wrote:sure, dont forgot being shot!??
or asteroid falling on someone's heads?
Lots of things cause death/heart attacks, but I'm talking about things we can measure,
pamojja wrote:The most people die of is either calcification of arteries or cancer at old age. The only test which measures calcification is imaging, not Lp(a). Your friend has no disease of calcification if he measures it. Lp(a) doesn't measures calcification.
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