Moderator: ofonorow
ofonorow wrote:
I don't know. What I think I know is that unlike lysine (which is essential, meaning all our lysine comes from the diet) our bodies can make proline, and my pet "theory" is that as we age, we make less, like everything else, leading to more atherosclerosis.
So how do you determine the "right" amount of proline? I have been using Lp(a) because our experience tells us that proline is the factor the eventually leads to an almost zero Lp(a) measurement.
In the initial product, Tower Heart Technology, proline was something like 400 mg per serving. Most people needing a therapeutic dosage would take two servings of HT daily, or 800 mg of proline. These results were miraculous, but we really didn't measure or study Lp(a).
I know that some alternative doctors recommend 2000 mg (2 g) of proline, but I am not sure of the basis of this recommendation.
It would be nice to know the amount of proline in the blood, the turn-over, etc. For me, the higher my Lp(a), the more proline I would consider.
ofonorow wrote:First, vitamin E is very important, especially in someone who has elevated Lp(a) (risk factor).
My guess is that you probably require more vitamin C given that 10 grams creates no reaction. The Jaffe Calibration/Cleanse, i.e. the highest oral one-time amount every 15 minutes until the watery discharge, might be worthwhile to gauge how much vitamin C your are able to metabolize. (If you try that, let us know how much C you had to take.)
Assuming the 57 Lp(a) score is measured, not calculated, then taking Lp(a) binding inhibitors (e.g. vitamin C, lysine and proline) make a lot of sense, whether or not the Lp(a) being produced declines.
One other thing is that the danger of 57 mg/dl depends on how many and the size of the Lp(a) particles. The size lf Lp(a) is not uniform. This is probably the reason that they changed to particles (e.g. nmol/l) rather than weight (mg/dl). A fewer number of large molecules are not as much risk as a larger number of smaller Lp(a) particles - though they both might have the same weight.
Atherotech (apparently now defunct) solved this by reporting Lp(a) as the weight of the same number of ordinary LDL particles.
Are you taking any drugs?
Frodo wrote:
Thank you, Owen
Despite my mci last year, I don't take all the drugs. Except baby aspirin (every second day 35 mg). I want to cut it too, next month. And (look at johnwens answer) I take indeed l-thyrox. Of course I regularly take vitamin E (800 IU). And the lab has really measured nmol lp(a). Value is 137 nmol.
francisunderwood wrote:Frodo wrote:
Thank you, Owen
Despite my mci last year, I don't take all the drugs. Except baby aspirin (every second day 35 mg). I want to cut it too, next month. And (look at johnwens answer) I take indeed l-thyrox. Of course I regularly take vitamin E (800 IU). And the lab has really measured nmol lp(a). Value is 137 nmol.
What type of Vitamin E do you take?
Frodo wrote:
Vitamin E complex from Greenfood and tocotrienols from Now.
Frodo wrote:Owen, please look at post 40. Do you think it could be caused by the medications (aspirin and thyroxin)?
Lp(a) didn't decline further until now. OK. I think it needs time. But there are other good news: Ejection fraction has increased to 55-60 %. And the part of my heartmuscle, that was damaged by mci, has started to work, unexpected. I'm happy.
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